Hepcidin and HFE protein: Iron metabolism as a target for the anemia of chronic kidney disease

doi:10.5527/wjn.v1.i6.166

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Dec 6, 2012 (publication date) through Dec 25, 2024

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World Journal of Nephrology

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2220-6124

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Baishideng Publishing Group Inc, 7041 Koll Center Parkway, Suite 160, Pleasanton, CA 94566, USA

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World J Nephrol. Dec 6, 2012; 1(6): 166-176
Published online Dec 6, 2012. doi: 10.5527/wjn.v1.i6.166

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Figure 2 Impact of HFE mutations on erythropoiesis in chronic kidney disease. EPO: Erythropoietin, Epo-R: Erythropoietin receptor; TFR1: Transferrin receptor 1; TFR2: Transferrin receptor 2; HFE: Hereditary hemochromatosis protein; TSAT: Transferrin saturation; Hb: Hemoglobin.

Citation: Canavesi E, Alfieri C, Pelusi S, Valenti L. Hepcidin and HFE protein: Iron metabolism as a target for the anemia of chronic kidney disease. World J Nephrol 2012; 1(6): 166-176
URL: https://www.wjgnet.com/2220-6124/full/v1/i6/166.htm
DOI: https://dx.doi.org/10.5527/wjn.v1.i6.166