Hepcidin and HFE protein: Iron metabolism as a target for the anemia of chronic kidney disease

doi:10.5527/wjn.v1.i6.166

Advanced Search

BPG is committed to discovery and dissemination of knowledge

Home / Archive / Volume 1, Issue 6

This Article

Citation of this article

Corresponding Author of This Article

Article-Type of This Article

Open-Access Policy of This Article

Times Cited Counts in Google of This Article

Number of Hits and Downloads for This Article

Total Article Views (9107)

All Articles published online

The chart showing PDF series, HTML series, Figures (1-3) series, Tables (1-1) series.

Item

Count

PDF

597

HTML

7841

Figures (1-3)

385

Tables (1-1)

284

Sum=9107

Dec 6, 2012 (publication date) through Nov 22, 2024

Times Cited of This Article

Times Cited (27)

Journal Information of This Article

Publication Name

World Journal of Nephrology

ISSN

2220-6124

Publisher of This Article

Baishideng Publishing Group Inc, 7041 Koll Center Parkway, Suite 160, Pleasanton, CA 94566, USA

Review

World J Nephrol. Dec 6, 2012; 1(6): 166-176
Published online Dec 6, 2012. doi: 10.5527/wjn.v1.i6.166

Full Size Figure Download Figure

Figure 2 Impact of HFE mutations on erythropoiesis in chronic kidney disease. EPO: Erythropoietin, Epo-R: Erythropoietin receptor; TFR1: Transferrin receptor 1; TFR2: Transferrin receptor 2; HFE: Hereditary hemochromatosis protein; TSAT: Transferrin saturation; Hb: Hemoglobin.

Citation: Canavesi E, Alfieri C, Pelusi S, Valenti L. Hepcidin and HFE protein: Iron metabolism as a target for the anemia of chronic kidney disease. World J Nephrol 2012; 1(6): 166-176
URL: https://www.wjgnet.com/2220-6124/full/v1/i6/166.htm
DOI: https://dx.doi.org/10.5527/wjn.v1.i6.166