How virus persistence can initiate the tumorigenesis process

doi:10.5501/wjv.v2.i2.102

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May 12, 2013 (publication date) through Jul 17, 2024

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World Journal of Virology

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2220-3249

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Baishideng Publishing Group Inc, 7041 Koll Center Parkway, Suite 160, Pleasanton, CA 94566, USA

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World J Virol. May 12, 2013; 2(2): 102-109
Published online May 12, 2013. doi: 10.5501/wjv.v2.i2.102

Table 3 Virus products controlling cellular epigenetic modifications

Virus	EBV	HHV-8	HPV	HBV	HCV	HTLV-1
	EBNA-3A, EBNA-3C[78]	LANA-1[81]	E6[83]	HBx[85,86]	Core[87]	Tax[88]
	LMP-1[79]	microRNA[82]	E7[84]
	LMP-2[80]

EBV: Epstein-Barr virus; HHV-8: Human herpesvirus 8, also named Kaposi sarcoma virus; HPV: Human papillomavirus; HBV: Hepatatis B virus; HCV: Hepatitis C virus; HTLV-1: Human T-cell leukemia virus 1.

Citation: Avanzi S, Alvisi G, Ripalti A. How virus persistence can initiate the tumorigenesis process. World J Virol 2013; 2(2): 102-109
URL: https://www.wjgnet.com/2220-3249/full/v2/i2/102.htm
DOI: https://dx.doi.org/10.5501/wjv.v2.i2.102