Complement-mediated renal diseases after kidney transplantation - current diagnostic and therapeutic options in de novo and recurrent diseases

doi:10.5500/wjt.v8.i6.203

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Oct 22, 2018 (publication date) through Dec 17, 2024

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World Journal of Transplantation

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2220-3230

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Baishideng Publishing Group Inc, 7041 Koll Center Parkway, Suite 160, Pleasanton, CA 94566, USA

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World J Transplantation. Oct 22, 2018; 8(6): 203-219
Published online Oct 22, 2018. doi: 10.5500/wjt.v8.i6.203

Table 4 Overview of mutations in complement factor H-related protein genes

Genetic defect	Phenotypical expression
Duplication in CFHR5 gene	C3 glomerulopathy (CFHR5 nephropathy)
Duplication in CFHR1 gene	C3 glomerulopathy
Hybrid CFHR3/CFHR1	C3 glomerulopathy
Hybrid CFHR2/CFHR5	C3 glomerulopathy
Hybrid CFH/CFHR1	aHUS
Hybrid CFH/CFHR3	aHUS

Adapted from Salvadori et al[1]. aHUS: Atypical hemolytic uremic syndrome; CFH: Complement factor H.

Citation: Abbas F, El Kossi M, Kim JJ, Shaheen IS, Sharma A, Halawa A. Complement-mediated renal diseases after kidney transplantation - current diagnostic and therapeutic options in de novo and recurrent diseases. World J Transplantation 2018; 8(6): 203-219
URL: https://www.wjgnet.com/2220-3230/full/v8/i6/203.htm
DOI: https://dx.doi.org/10.5500/wjt.v8.i6.203