Pharmacological Tie2 activation in kidney transplantation

doi:10.5500/wjt.v6.i3.573

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World Journal of Transplantation

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2220-3230

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Baishideng Publishing Group Inc, 7041 Koll Center Parkway, Suite 160, Pleasanton, CA 94566, USA

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World J Transplant. Sep 24, 2016; 6(3): 573-582
Published online Sep 24, 2016. doi: 10.5500/wjt.v6.i3.573

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Figure 4 Vasculotide ameliorates fibrogenesis. Fluorescent immunohistochemistry of α-smooth-muscle-actin (red) regarding (A) tubular and (B) glomerular injury in kidney cross-sections of transplanted mice (vehicle or VT-treated) on day 28 after transplantation. Autofluorescence is shown in green. Images are exemplary for n = 5/condition; C: Sirius red staining for the same conditions (D) Immunoblot of murine kidney homogenates for pSMAD3 in healthy (day 0) and kidney transplanted mice (day 28) upon vehicle or VT treatment. VT: Vasculotide; αSMA: α-smooth-muscle-actin; GAPDH: Glyceraldehyde 3-phosphate dehydrogenase.

Citation: Thamm K, Njau F, Van Slyke P, Dumont DJ, Park JK, Haller H, David S. Pharmacological Tie2 activation in kidney transplantation. World J Transplant 2016; 6(3): 573-582
URL: https://www.wjgnet.com/2220-3230/full/v6/i3/573.htm
DOI: https://dx.doi.org/10.5500/wjt.v6.i3.573