Thrombotic microangiopathy after kidney transplantation: Expanding etiologic and pathogenetic spectra

doi:10.5500/wjt.v14.i1.90277

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World Journal of Transplantation

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2220-3230

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Baishideng Publishing Group Inc, 7041 Koll Center Parkway, Suite 160, Pleasanton, CA 94566, USA

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World J Transplant. Mar 18, 2024; 14(1): 90277
Published online Mar 18, 2024. doi: 10.5500/wjt.v14.i1.90277

Table 1 Etiology and classification of thrombotic microangiopathies

Primary TMAs
	Shiga toxin-producing E. Coli-associated HUS
	Thrombotic thrombocytopenic purpura
	Atypical HUS or complement-mediated
Secondary TMAs
	Infections including viral, fungal, and bacterial
	Drugs including immunosuppressants and chemotherapeutic agents
	Autoimmune diseases
	Malignant hypertension
	Malignancy
	Metabolic defects
	Pregnancy
	Transplantation, both hematopoietic stem cell transplantation and solid organ transplantation
	Disseminated intravascular coagulation
	Radiation

TMAs: Thrombotic microangiopathies.

Citation: Mubarak M, Raza A, Rashid R, Sapna F, Shakeel S. Thrombotic microangiopathy after kidney transplantation: Expanding etiologic and pathogenetic spectra. World J Transplant 2024; 14(1): 90277
URL: https://www.wjgnet.com/2220-3230/full/v14/i1/90277.htm
DOI: https://dx.doi.org/10.5500/wjt.v14.i1.90277