Post-reperfusion syndrome in liver transplant recipients: What is new in prevention and management?

Table 1 Potential cellular and physiologic causes of post-reperfusion syndrome

Category	Physiologic change	Hemodynamic change
Release of vasoactive substances	Inflammatory cytokines (nitrous oxide, endothelin-1, bradykinin, reactive oxygen species)	Increased CVP; increased PVR; increased PAP; decreased SVR; decreased ABP; decreased CI; decreased HR; ST changes; T-wave changes
Electrolyte disturbances	Hypothermia; acidosis; hyperkalemia; hypocalcemia; hyperphosphatemia
Embolic phenomena	Thromboembolic; air emboli

ABP: Arterial blood pressure; CI: Cardiac index; CVP: Central venous pressure; HR: Heart rate; PAP: Pulmonary artery pressure; PVR: Pulmonary vascular resistance; SVR: Systemic vascular resistance.