Role of β-microseminoprotein from prostate cancer initiation to recurrence: A mini-review

Table 1 A summary of recorded functions of β-microsemi-noprotein

Growth inhibition and apoptosis control of prostate epithelium	[16]
Decrease of tumor growth, hypercalcemia and skeletal metastases	[17]
Decrease tumor vessel density	[19]
Anti-vascular endothelial growth factor effects	[19]
Inhibit the vascular endothelial growth factor binding to tyrosine kinase receptor Flk-1/KDR expressed by endothelial cells	[19]
Inhibits the secretion of a matrix metalloproteinase	[20]
Controls anchorage-independent colony growth of prostate cancer	[21]
Interaction with IgG and modulating immune function	[57]
Interaction with CRISP-3 and modulating ion channel regulation	[43]
Interaction with CRISP-3 to inhibit effects on innate immunity	[43]
A mediator of natural killer cells and an activator of neutrophils	[53]
A pH and Ca2+ dependent candidacidal activity	[23]

IgG: Immunoglobulin G; Flk-1/KDR: Fetal liver kinase 1/Kinase insert domain receptor; CRISP-3: Cysteine-rich secretory proteins.