Post-COVID-19 persistent olfactory, gustatory, and trigeminal chemosensory disorders: Definitions, mechanisms, and potential treatments

Figure 5 Mechanism of dysosmia after severe acute respiratory syndrome coronavirus 2 infection. A: The disorganized olfactory neuroepithelium after severe acute respiratory syndrome coronavirus 2 infection and its associated immunological response would result in a disturbance of rewiring of the glomeruli; B: A glomerulus is no longer dominated by a single type of odor and the newly growing axon after injury would go to a different brain spot other than the one before injury, resulting in a distorted sense of smell.