Apoptosis and endometrial receptivity: Relationship with in vitro fertilization treatment outcome

doi:10.5317/wjog.v5.i1.87

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Feb 10, 2016 (publication date) through Nov 4, 2024

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World Journal of Obstetrics and Gynecology

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2218-6220

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Baishideng Publishing Group Inc, 7041 Koll Center Parkway, Suite 160, Pleasanton, CA 94566, USA

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World J Obstet Gynecol. Feb 10, 2016; 5(1): 87-96
Published online Feb 10, 2016. doi: 10.5317/wjog.v5.i1.87

Table 1 Regulation of apoptosis in normal endometrium during menstrual cycle

Proliferative phase		Secretory phase
Increasedexpression	Inhibited expression	Increased expression	Inhibited expression
Bcl-2	p53	BAX	Bcl-2
Fas, FasL	PTEN	Fas, FasL
	PPARγ	TRAIL-R
	DFF45	PTEN
		Caspase-3
		PPARγ
		DFF 45

Bcl-2: B cell lymphoma/leukemia-2; PTEN: Phosphatase and tensin homolog; PPARγ: Peroxisome proliferator-activated receptor gamma; TRAIL: TNF-related apoptosis-inducing ligand; DFF45: DNA fragmentation factor of 45 kDa.

Citation: Antsiferova YS, Sotnikova NY. Apoptosis and endometrial receptivity: Relationship with in vitro fertilization treatment outcome. World J Obstet Gynecol 2016; 5(1): 87-96
URL: https://www.wjgnet.com/2218-6220/full/v5/i1/87.htm
DOI: https://dx.doi.org/10.5317/wjog.v5.i1.87