Influence of Helicobacter pylori oncoprotein CagA in gastric cancer: A critical-reflective analysis

doi:10.5306/wjco.v13.i11.866

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Nov 24, 2022 (publication date) through Nov 22, 2024

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World Journal of Clinical Oncology

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2218-4333

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Baishideng Publishing Group Inc, 7041 Koll Center Parkway, Suite 160, Pleasanton, CA 94566, USA

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World J Clin Oncol. Nov 24, 2022; 13(11): 866-879
Published online Nov 24, 2022. doi: 10.5306/wjco.v13.i11.866

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Figure 3 Status of the current understanding regarding CagA-related pathogenic mechanisms. EMT: Epithelial to mesenchymal transition; CEACAMs: Carcinoembryonic antigen-related cell adhesion molecules; Grb2: Growth factor receptor-bound protein 2; ASPP2: Apoptosis-stimulating protein of p53 2; SHP2: Domain-containing protein tyrosine phosphatase 2; SHIP2: Src homology 2 domain-containing inositol 5'-phosphatase 2; RUNX3: Runt-related transcription factor 3; PAR1: Partitioning-defective 1; NikS: Nickel-regulated sRNA.

Citation: Freire de Melo F, Marques HS, Rocha Pinheiro SL, Lemos FFB, Silva Luz M, Nayara Teixeira K, Souza CL, Oliveira MV. Influence of Helicobacter pylori oncoprotein CagA in gastric cancer: A critical-reflective analysis. World J Clin Oncol 2022; 13(11): 866-879
URL: https://www.wjgnet.com/2218-4333/full/v13/i11/866.htm
DOI: https://dx.doi.org/10.5306/wjco.v13.i11.866