Current understanding of the neuropathophysiology of pain in chronic pancreatitis

doi:10.4291/wjgp.v6.i4.193

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Nov 15, 2015 (publication date) through Nov 4, 2024

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World Journal of Gastrointestinal Pathophysiology

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2150-5330

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Baishideng Publishing Group Inc, 7041 Koll Center Parkway, Suite 160, Pleasanton, CA 94566, USA

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World J Gastrointest Pathophysiol. Nov 15, 2015; 6(4): 193-202
Published online Nov 15, 2015. doi: 10.4291/wjgp.v6.i4.193

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Figure 1 The mechanism of peripheral sensitization. PAR2: Proteinase-activated receptor 2; NGF: Nerve growth factor; TRPA1: Transient receptor potential ankyrin 1; TRPV1: Transient receptor potential vanilloid 1; BDNF: Brain-derived neurotrophic factor; GAP43: Growth-associated protein 43; LTB4: Leukotriene B4.

Citation: Atsawarungruangkit A, Pongprasobchai S. Current understanding of the neuropathophysiology of pain in chronic pancreatitis. World J Gastrointest Pathophysiol 2015; 6(4): 193-202
URL: https://www.wjgnet.com/2150-5330/full/v6/i4/193.htm
DOI: https://dx.doi.org/10.4291/wjgp.v6.i4.193