Prevention of post-ERCP pancreatitis

doi:10.4291/wjgp.v5.i1.1

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Feb 15, 2014 (publication date) through Jul 2, 2024

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World Journal of Gastrointestinal Pathophysiology

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2150-5330

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Baishideng Publishing Group Inc, 7041 Koll Center Parkway, Suite 160, Pleasanton, CA 94566, USA

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World J Gastrointest Pathophysiol. Feb 15, 2014; 5(1): 1-10
Published online Feb 15, 2014. doi: 10.4291/wjgp.v5.i1.1

Table 3 Pharmacological agents studied according to postulated mechanism of action

Postulated mechanismof action	Agents
Interruption of inflam matory cascade	NSAIDs, steroids, interleukin-10, allopurinol, adrenaline spray, pentoxifylline, platelet-activating factor-acetylhidrolase, semapimod, aprepitant, risperidone
Reduction of pancreatic enzyme secretion	Octreotide, somatostatin, calcitonin
Inhibition of protease activity	Gabexate mesilate, heparin, ulinastatin, nafamostat, magnesium sulphate
Reduction of Sphincter- of-Oddi pressure	Nitroglycerin, nifedipine, botulinum toxin, lidocaine, secretin, phosphodiesterase inhibitor type 5
Prevention of infection	Antibiotics
Anti-oxidants	Beta-carotene, N-acetylcysteine, sodium selenite
Anti-metabolites	5-fluorouracil

NSAIDs: Non-steroidal anti-inflammatory drugs.

Citation: Wong LL, Tsai HH. Prevention of post-ERCP pancreatitis. World J Gastrointest Pathophysiol 2014; 5(1): 1-10
URL: https://www.wjgnet.com/2150-5330/full/v5/i1/1.htm
DOI: https://dx.doi.org/10.4291/wjgp.v5.i1.1