Histamine 3 receptor activation mediates inhibition of acid secretion during Helicobacter-induced gastritis

doi:10.4291/wjgp.v1.i5.154

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Dec 15, 2010 (publication date) through Nov 12, 2024

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World Journal of Gastrointestinal Pathophysiology

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2150-5330

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Baishideng Publishing Group Inc, 7041 Koll Center Parkway, Suite 160, Pleasanton, CA 94566, USA

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World J Gastrointest Pathophysiol. Dec 15, 2010; 1(5): 154-165
Published online Dec 15, 2010. doi: 10.4291/wjgp.v1.i5.154

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Figure 9 Proposed role for the H3R in gastric function during Helicobacter infection. A: H3Rs localized on parietal cells and D-cells supports a direct inhibitory effect of the H. pylori metabolite Nα-MH on acid and somatostatin secretion respectively during infection; B: The stimulatory effect of thioperamide on somatostatin expression and secretion regulates the host immune response during H. pylori infection; H3R: histamine 3 receptor; SOM: somatostatin.

Citation: Zavros Y, Mesiwala N, Waghray M, Todisco A, Shulkes A, Merchant JL. Histamine 3 receptor activation mediates inhibition of acid secretion during Helicobacter-induced gastritis. World J Gastrointest Pathophysiol 2010; 1(5): 154-165
URL: https://www.wjgnet.com/2150-5330/full/v1/i5/154.htm
DOI: https://dx.doi.org/10.4291/wjgp.v1.i5.154