Metabolic syndrome in hypertensive patients: An unholy alliance

doi:10.4330/wjc.v6.i9.890

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Sep 26, 2014 (publication date) through Dec 25, 2024

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World Journal of Cardiology

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1949-8462

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Baishideng Publishing Group Inc, 7041 Koll Center Parkway, Suite 160, Pleasanton, CA 94566, USA

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World J Cardiol. Sep 26, 2014; 6(9): 890-907
Published online Sep 26, 2014. doi: 10.4330/wjc.v6.i9.890

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Figure 1 Hypothetical mechanisms by which the metabolic syndrome may lead to high blood pressure. NO: Nitric oxide; ET-1: Endothelin-1; SNS: Sympathetic nervous system; RAAS: Renin-angiotensin-aldosterone system; CTRP1: Complement-C1q tumor necrosis factor-related protein 1; EKODE: Epoxy-keno derivative of linoleic acid; TPR: Total peripheral resistance; EDLF: Endogenous digoxin-like factor; [Ca_i²⁺]: Intracellular concentration of calcium; [Na_i⁺]: Intracellular concentration of sodium.

Citation: Mulè G, Calcaterra I, Nardi E, Cerasola G, Cottone S. Metabolic syndrome in hypertensive patients: An unholy alliance. World J Cardiol 2014; 6(9): 890-907
URL: https://www.wjgnet.com/1949-8462/full/v6/i9/890.htm
DOI: https://dx.doi.org/10.4330/wjc.v6.i9.890