Adenosine triphosphate-induced cell death in heart failure: Is there a link?

doi:10.4330/wjc.v17.i4.105021

Advanced Search

BPG is committed to discovery and dissemination of knowledge

Home / Archive / Volume 17, Issue 4

This Article

Academic Content and Language Evaluation of This Article

CrossCheck and Google Search of This Article

Academic Rules and Norms of This Article

Citation of this article

Corresponding Author of This Article

Research Domain of This Article

Article-Type of This Article

Open-Access Policy of This Article

Times Cited Counts in Google of This Article

Number of Hits and Downloads for This Article

Total Article Views (25)

All Articles published online

The chart showing PDF series, HTML series, Figures (1-4) series, Tables (1-3) series.

Item

Count

PDF

HTML

Figures (1-4)

Tables (1-3)

Sum=24

Apr 26, 2025 (publication date) through Apr 21, 2025

Times Cited of This Article

Times Cited (0)

Journal Information of This Article

Publication Name

World Journal of Cardiology

ISSN

1949-8462

Publisher of This Article

Baishideng Publishing Group Inc, 7041 Koll Center Parkway, Suite 160, Pleasanton, CA 94566, USA

Review

World J Cardiol. Apr 26, 2025; 17(4): 105021
Published online Apr 26, 2025. doi: 10.4330/wjc.v17.i4.105021

Full Size Figure Download Figure

Figure 4 Adenosine triphosphate causes the loss of mitochondrial membrane potential, the disruption of mitochondrial integrity, the production of reactive oxygen species and alterations in mitochondrial membrane permeability, collectively leading to cell deaths. ATP: Adenosine triphosphate; P2RX: Purinoceptor receptor P2X; PANX1: Pannexin 1; DNA: Deoxyribonucleic acid; ROS: Reactive oxygen species; mPTP: Mitochondrial permeability transition pore. Drawn by Figdraw.

Citation: Zhang JJ, Cheng L, Qiao Q, Xiao XL, Lin SJ, He YF, Sha RL, Sha J, Ma Y, Zhang HL, Ye XR. Adenosine triphosphate-induced cell death in heart failure: Is there a link? World J Cardiol 2025; 17(4): 105021
URL: https://www.wjgnet.com/1949-8462/full/v17/i4/105021.htm
DOI: https://dx.doi.org/10.4330/wjc.v17.i4.105021