Endocrine disrupting chemicals in mixture and obesity, diabetes and related metabolic disorders

doi:10.4331/wjbc.v8.i2.108

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World Journal of Biological Chemistry

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1949-8454

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Baishideng Publishing Group Inc, 7041 Koll Center Parkway, Suite 160, Pleasanton, CA 94566, USA

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World J Biol Chem. May 26, 2017; 8(2): 108-119
Published online May 26, 2017. doi: 10.4331/wjbc.v8.i2.108

Table 2 Metabolic characteristics of mice deficient in some nuclear receptors¹

Insulin status	Obesity	No body weight change
Insulin resistance	ERα (-/-) in both males and females[38]
No difference in insulin sensitivity	AR (-/-) in males only[97]
Improved insulin sensitivity	ERβ (-/-) (study on males only)[26]	CAR activation (study on males only in HFD context, activation by TOBOBOP)[99]
	ERRβ (deletion in neurons; study on males only)[98]	AhR (-/-) (studies on males only)[100]
		AhR (-/-) (studies on males only, in HFD context)[101]
		PPARα (-/-) (studies on males only, in HFD context)[102]
		PXR (-/-) (studies on males only, in HFD context)[103]

¹Mice were fed standard diet or high-fat diet when mentioned. HFD: High-fat diet; AhR: Aryl hydrocarbon receptor; CAR: Constitutive androstane receptor; PPARα: Peroxisome proliferator-activated receptor α; PXR: Pregnane X receptor.

Citation: Le Magueresse-Battistoni B, Labaronne E, Vidal H, Naville D. Endocrine disrupting chemicals in mixture and obesity, diabetes and related metabolic disorders. World J Biol Chem 2017; 8(2): 108-119
URL: https://www.wjgnet.com/1949-8454/full/v8/i2/108.htm
DOI: https://dx.doi.org/10.4331/wjbc.v8.i2.108