Copyright
©The Author(s) 2015.
World J Diabetes. Jul 10, 2015; 6(7): 943-960
Published online Jul 10, 2015. doi: 10.4239/wjd.v6.i7.943
Published online Jul 10, 2015. doi: 10.4239/wjd.v6.i7.943
Figure 3 Illustration of the cardiac sarcomere indicating the location of actin thin-filament and myosin thick-filament accessory proteins involved in the regulation of actin-myosin cross-bridge dynamics and kinetics.
C0-C10: Immunoglobulin-like and fibronectin-like domains of myosin binding protein C; MD: M-domain of myosin binding protein C containing protein kinase phosphorylation sites.
- Citation: Waddingham MT, Edgley AJ, Tsuchimochi H, Kelly DJ, Shirai M, Pearson JT. Contractile apparatus dysfunction early in the pathophysiology of diabetic cardiomyopathy. World J Diabetes 2015; 6(7): 943-960
- URL: https://www.wjgnet.com/1948-9358/full/v6/i7/943.htm
- DOI: https://dx.doi.org/10.4239/wjd.v6.i7.943