Molecular mechanisms of protein induced hyperinsulinaemic hypoglycaemia

doi:10.4239/wjd.v5.i5.666

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Oct 15, 2014 (publication date) through Nov 25, 2024

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World Journal of Diabetes

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1948-9358

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Baishideng Publishing Group Inc, 7041 Koll Center Parkway, Suite 160, Pleasanton, CA 94566, USA

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World J Diabetes. Oct 15, 2014; 5(5): 666-677
Published online Oct 15, 2014. doi: 10.4239/wjd.v5.i5.666

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Figure 4 β-oxidation of fatty acids. Acyl-CoA is converted to acetyl-CoA through dehydrogenation, hydration, oxidation and thiolysis. Acetyl-CoA can enter the Krebs cycle or can lead to ketogenesis. CPT-1: Carnitine palmitoyltransferase-1; SCHAD: 3-hydroxyacyl CoA dehydrogenase.

Citation: Chandran S, Yap F, Hussain K. Molecular mechanisms of protein induced hyperinsulinaemic hypoglycaemia. World J Diabetes 2014; 5(5): 666-677
URL: https://www.wjgnet.com/1948-9358/full/v5/i5/666.htm
DOI: https://dx.doi.org/10.4239/wjd.v5.i5.666