Neurophysiological mechanisms of electroacupuncture in regulating pancreatic function and adipose tissue expansion

Figure 2 Electroacupuncture modulates inflammatory mediators in peripancreatic adipose tissue, enhancing metabolic function. A and B: Electroacupuncture (EA)'s impact on the expression of F4/80 (also known as EGF-like module-containing mucin-like hormone receptor-like 1); A and C: Influence of EA on MCP-1 levels; A and D: Changes in interleukin (IL)-1β expression due to EA treatment; A and E: EA's effect on IL-10 expression; F and G: Expression levels of PPAR-γ following EA intervention; F and H: Adipose tissue expression of FABP4 in response to EA; F and I: TRPV1 expression in PAT after EA treatment. Vinculin served as a loading control; J: Representative immunofluorescence images of co-expression of FABP4 and TRPV1 in the pancreas of the model group (200 × magnification). Blue fluorescence representing FABP4, and TRPV1 is represented by purple fluorescence; K: Quantitative analysis of the co-localization of FABP4 and TRPV1 immunofluorescence, indicating a correlation between the two proteins. ^aP < 0.05 compared to the normal control group; ^bP < 0.05 compared to the model or another treatment group.