MiR-29a-3p inhibits fibrosis of diabetic kidney disease in diabetic mice via downregulation of DNA methyl transferase 3A and 3B

doi:10.4239/wjd.v16.i4.93630

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World Journal of Diabetes

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1948-9358

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Baishideng Publishing Group Inc, 7041 Koll Center Parkway, Suite 160, Pleasanton, CA 94566, USA

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World J Diabetes. Apr 15, 2025; 16(4): 93630
Published online Apr 15, 2025. doi: 10.4239/wjd.v16.i4.93630

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Figure 8 Change in the expression of Wnt3a/β-catenin and Janus kinase/signal transducer and activator of transcription pathways in the diabetic mice with agomiR-29a-3p treatment. The expression of Wnt3a, β-catenin, Janus kinase (JAK) 2, and signal transducer and activator of transcription (Stat) 3 in the diabetic (agomiR-29a-3p) mice were significantly lower than that in the diabetic mice without agomiR-29a-3p treatment (n = 3). CON: Control group; NC: Negative control.

Citation: Yang Y, Chen Y, Tang JY, Chen J, Li GQ, Feng B, Mu J. MiR-29a-3p inhibits fibrosis of diabetic kidney disease in diabetic mice via downregulation of DNA methyl transferase 3A and 3B. World J Diabetes 2025; 16(4): 93630
URL: https://www.wjgnet.com/1948-9358/full/v16/i4/93630.htm
DOI: https://dx.doi.org/10.4239/wjd.v16.i4.93630