cNPAS2 induced β cell dysfunction by regulating KANK1 expression in type 2 diabetes

doi:10.4239/wjd.v15.i9.1932

Advanced Search

BPG is committed to discovery and dissemination of knowledge

Home / Archive / Volume 15, Issue 9

This Article

Academic Content and Language Evaluation of This Article

CrossCheck and Google Search of This Article

Academic Rules and Norms of This Article

Citation of this article

Corresponding Author of This Article

Research Domain of This Article

Article-Type of This Article

Open-Access Policy of This Article

Times Cited Counts in Google of This Article

Number of Hits and Downloads for This Article

Total Article Views (788)

All Articles published online

The chart showing PDF series, WORD series, HTML series, Figures (1-7) series, Tables (1-1) series.

Item

Count

PDF

WORD

HTML

466

Figures (1-7)

Tables (1-1)

Sum=663

Publishing Process of This Article

The chart showing Browse series, Download series.

Item

Count

Browse

Download

Sum=81

Sep 15, 2024 (publication date) through Nov 5, 2024

Times Cited of This Article

Times Cited (0)

Journal Information of This Article

Publication Name

World Journal of Diabetes

ISSN

1948-9358

Publisher of This Article

Baishideng Publishing Group Inc, 7041 Koll Center Parkway, Suite 160, Pleasanton, CA 94566, USA

Basic Study

World J Diabetes. Sep 15, 2024; 15(9): 1932-1941
Published online Sep 15, 2024. doi: 10.4239/wjd.v15.i9.1932

Full Size Figure Download Figure

Figure 3 KANK1 was the potential target gene of NPAS2. A: Scatter plot revealed the favorable connection between KANK1 and NPAS2 in diabetes mellitus type 2 (T2DM); B and C: The expression of KANK1 in T2DM group was higher than that in control group. ^aP < 0.0001. T2DM: Diabetes mellitus type 2.

Citation: Yin YB, Ji W, Liu YL, Gao QH, He DD, Xu SL, Fan JX, Zhang LH. cNPAS2 induced β cell dysfunction by regulating KANK1 expression in type 2 diabetes. World J Diabetes 2024; 15(9): 1932-1941
URL: https://www.wjgnet.com/1948-9358/full/v15/i9/1932.htm
DOI: https://dx.doi.org/10.4239/wjd.v15.i9.1932