Duodenal-jejunal bypass improves hypothalamic oxidative stress and inflammation in diabetic rats via glucagon-like peptide 1-mediated Nrf2/HO-1 signaling

Figure 1 The expression of differential proteins in the hypothalamus after duodenal jejunal bypass intervention. A: Heat map of the expression of the 120 DEPs between the T [type 2 diabetes mellitus (T2DM)-Sham] rats and D [T2DM-duodenal jejunal bypass (DJB)] rats. The colored column represents the sample number, the row name indicates the DEGs, each rectangle in the graph corresponds to the expression value of a sample, red indicates high expression and blue indicates low expression; B: Statistics of significantly enriched Kyoto encyclopedia of genes and genomes pathways (T2DM-Sham vs T2DM-DJB); C: GO term statistics for significantly enriched genes (T2DM-Sham vs T2DM-DJB).