Shikonin protects mitochondria through the NFAT5/AMPK pathway for the treatment of diabetic wounds

doi:10.4239/wjd.v15.i12.2338

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Dec 15, 2024 (publication date) through Nov 20, 2024

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World Journal of Diabetes

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1948-9358

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Baishideng Publishing Group Inc, 7041 Koll Center Parkway, Suite 160, Pleasanton, CA 94566, USA

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World J Diabetes. Dec 15, 2024; 15(12): 2338-2352
Published online Dec 15, 2024. doi: 10.4239/wjd.v15.i12.2338

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Figure 3 Effects of shikonin on blood glucose, wound healing, and NFAT5 expression in diabetic mice. A: Changes of blood glucose in mice (D3 vs D1, P < 0.05); B: Wound changes in shikonin group and blank group. Shikonin group after modeling: Day 1, day 3, day 6. Blank group after modeling: day 1, day 3, day 6; C: Wound area changing and wound healing time; D: The immunofluorescence expressions of NFAT5 protein of diabetic mice from wound margin. NFAT5 antibody: red, cell nucleus: Blue; E: The expressions of NFAT5 protein of mice from wound margin. ^aP < 0.05 Shikonin group vs blank group.

Citation: Cen LS, Cao Y, Zhou YM, Guo J, Xue JW. Shikonin protects mitochondria through the NFAT5/AMPK pathway for the treatment of diabetic wounds. World J Diabetes 2024; 15(12): 2338-2352
URL: https://www.wjgnet.com/1948-9358/full/v15/i12/2338.htm
DOI: https://dx.doi.org/10.4239/wjd.v15.i12.2338