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©The Author(s) 2022.
World J Diabetes. Apr 15, 2022; 13(4): 338-357
Published online Apr 15, 2022. doi: 10.4239/wjd.v13.i4.338
Published online Apr 15, 2022. doi: 10.4239/wjd.v13.i4.338
Figure 7 Effects of SAR7334 and KN-93 on high-glucose-induced inhibition of H9C2 cell proliferation.
A: Percentage of cells in each group in each period was detected by flow cytometry. n = 5, unpaired t-test, aP < 0.001, bP < 0.01; B: Effects of SAR7334 and KN-93 on the expression levels of cycle related proteins PCNA and CyclinD1 in H9C2 cells induced by high glucose. n = 5, unpaired t-test, aP < 0.001, bP < 0.01, cP < 0.0001. n.s.: No statistical difference; C and D: Expression of PCNA detected by immunofluorescence. n = 5, unpaired t-test, aP < 0.001, bP < 0.01, cP < 0.0001. Scale bars = 10 μm. HG: High-glucose; LG: Low-glucose.
- Citation: Jiang SJ. Roles of transient receptor potential channel 6 in glucose-induced cardiomyocyte injury . World J Diabetes 2022; 13(4): 338-357
- URL: https://www.wjgnet.com/1948-9358/full/v13/i4/338.htm
- DOI: https://dx.doi.org/10.4239/wjd.v13.i4.338