Metformin regulates inflammation and fibrosis in diabetic kidney disease through TNC/TLR4/NF-κB/miR-155-5p inflammatory loop

doi:10.4239/wjd.v12.i1.19

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Jan 15, 2021 (publication date) through Nov 5, 2024

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World Journal of Diabetes

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1948-9358

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Baishideng Publishing Group Inc, 7041 Koll Center Parkway, Suite 160, Pleasanton, CA 94566, USA

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World J Diabetes. Jan 15, 2021; 12(1): 19-46
Published online Jan 15, 2021. doi: 10.4239/wjd.v12.i1.19

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Figure 2 Histological changes and tenascin-C expression in the kidneys of diabetic rats. A: Hematoxylin-eosin staining; B: Periodic acid-Schiff staining (400 ×); C: Immunohistochemical staining; D: Pathological scoring of tenascin-C (TNC). Scale bar: 20 μm. Representative immunohistochemical images and immunohistochemical scores for TNC in renal section are shown. ^aP < 0.05 compared with the NC control group. NC, n = 6, DN, n = 6. NC: Normal rat renal section; DN: Diabetic rat renal section; HE: Hematoxylin-eosin; PAS: Periodic acid-Schiff; TNC: Tenascin-C.

Citation: Zhou Y, Ma XY, Han JY, Yang M, Lv C, Shao Y, Wang YL, Kang JY, Wang QY. Metformin regulates inflammation and fibrosis in diabetic kidney disease through TNC/TLR4/NF-κB/miR-155-5p inflammatory loop. World J Diabetes 2021; 12(1): 19-46
URL: https://www.wjgnet.com/1948-9358/full/v12/i1/19.htm
DOI: https://dx.doi.org/10.4239/wjd.v12.i1.19