METTL5 promotes gastric cancer progression via sphingomyelin metabolism

doi:10.4251/wjgo.v16.i5.1925

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May 15, 2024 (publication date) through Jan 10, 2025

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World Journal of Gastrointestinal Oncology

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1948-5204

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Baishideng Publishing Group Inc, 7041 Koll Center Parkway, Suite 160, Pleasanton, CA 94566, USA

Clinical and Translational Research

World J Gastrointest Oncol. May 15, 2024; 16(5): 1925-1946
Published online May 15, 2024. doi: 10.4251/wjgo.v16.i5.1925

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Figure 8 METTL5 blunted the sensitivity of gastric cancer cells to cisplatin. A: Clone formation assay to detect the effect of cisplatin on the clone formation ability of gastric cancer cells after METTL5 knockdown; B: Transwell assay to detect the effect of cisplatin on the migration ability of gastric cancer cells after METTL5 knockdown. ^aP < 0.05, ^bP < 0.01, ^cP < 0.001.

Citation: Zhang YQ, Li J, Qin Z, Li DM, Ye FZ, Bei SH, Zhang XH, Feng L. METTL5 promotes gastric cancer progression via sphingomyelin metabolism. World J Gastrointest Oncol 2024; 16(5): 1925-1946
URL: https://www.wjgnet.com/1948-5204/full/v16/i5/1925.htm
DOI: https://dx.doi.org/10.4251/wjgo.v16.i5.1925