CALD1 facilitates epithelial-mesenchymal transition progression in gastric cancer cells by modulating the PI3K-Akt pathway

Figure 5 Impact of CALD1-shRNA on tumor growth and metastasis-related protein expression: Results from animal experiments. A: The mean weight of transplanted tumours in the CALD1-shRNA-transfected group was significantly lower than that in the empty vector-transfected group (P < 0.05), with a delayed growth curve and smaller final subcutaneous tumours; B: Compared with the empty vector-transfected group, the expression of N-cadherin, Dickkop-1, and Vimentin was reduced, whereas that of Claudin-1 and E-cadherin expression increased. ^bP < 0.01.