Pomolic acid and its glucopyranose ester promote apoptosis through autophagy in HT-29 colon cancer cells

Figure 1 The effects of pomolic acid and pomolic acid-28-O-β-D-glucopyranosyl ester on proliferation of colon cancer cells. A: The proliferation of HT-29 cells after treatment with different concentrations of pomolic acid (PA) for 24, 48, and 72 h; B: The proliferation of HT-29 cells after treatment with different concentrations of pomolic acid-28-O-β-D-glucopyranosyl ester (PAO) for 24, 48, and 72 h; C: The cell cycle distribution of HT-29 cells after treatment with different concentrations of PA for 24 h; D: The cell cycle distribution of HT-29 cells after treatment with different concentrations of PA for 48 h; E: The cell cycle distribution of HT-29 cells after treatment with different concentrations of PAO for 24 h; F: The cell cycle distribution of HT-29 cells after treatment with different concentrations of PAO for 48 h. ^aP < 0.05, ^bP < 0.01, ^cP < 0.001, ^dP < 0.0001.