Diabetes mellitus contribution to the remodeling of the tumor microenvironment in gastric cancer

doi:10.4251/wjgo.v13.i12.1997

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World Journal of Gastrointestinal Oncology

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1948-5204

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Baishideng Publishing Group Inc, 7041 Koll Center Parkway, Suite 160, Pleasanton, CA 94566, USA

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World J Gastrointest Oncol. Dec 15, 2021; 13(12): 1997-2012
Published online Dec 15, 2021. doi: 10.4251/wjgo.v13.i12.1997

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Figure 1 Diabetes mellitus can sculpture the tumor microenvironment of gastric cancer through a myriad of different molecular mechanisms ranging from dysregulation of cellular signaling pathways to marked metabolic disturbances. TME: Tumor microenvironment; ECM: Extracellular matrix; miRNAs: MicroRNAs; CAFs: Cancer-associated fibroblasts; TAMCs: Tumor-associated mast cells; LOX: Lysyl oxidase; TGF-β: Transforming growth factor β; RAGE: Receptor of advanced glycation end products; AGE: Advanced glycation end product; ChREBP: Carbohydrate-responsive element-binding protein.

Citation: Rojas A, Lindner C, Schneider I, Gonzàlez I, Araya H, Morales E, Gómez M, Urdaneta N, Araya P, Morales MA. Diabetes mellitus contribution to the remodeling of the tumor microenvironment in gastric cancer. World J Gastrointest Oncol 2021; 13(12): 1997-2012
URL: https://www.wjgnet.com/1948-5204/full/v13/i12/1997.htm
DOI: https://dx.doi.org/10.4251/wjgo.v13.i12.1997