PI3K/SHIP2/PTEN pathway in cell polarity and hepatitis C virus pathogenesis

doi:10.4254/wjh.v9.i1.18

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World Journal of Hepatology

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1948-5182

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Baishideng Publishing Group Inc, 7041 Koll Center Parkway, Suite 160, Pleasanton, CA 94566, USA

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World J Hepatol. Jan 8, 2017; 9(1): 18-29
Published online Jan 8, 2017. doi: 10.4254/wjh.v9.i1.18

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Figure 1 Phosphorylation and dephosphorylation cycles of different phosphoinositides. Monophosphate, diphosphate and triphosphate phosphoinositides are represented, as are the most widely studied kinases (in blue) and phosphatases (in red) that regulate their expression. PtdIns: Phosphatidylinositol; PI3K: Phosphoinositide 3-kinase; SHIP2: SH2-containing inositol polyphosphate 5-phosphatase; PTEN: Phosphatase and tensin homologue deleted on chromosome 10; OCRL: Inositol polyphosphate 5-phosphatase.

Citation: Awad A, Gassama-Diagne A. PI3K/SHIP2/PTEN pathway in cell polarity and hepatitis C virus pathogenesis. World J Hepatol 2017; 9(1): 18-29
URL: https://www.wjgnet.com/1948-5182/full/v9/i1/18.htm
DOI: https://dx.doi.org/10.4254/wjh.v9.i1.18