CD36 genetic variation, fat intake and liver fibrosis in chronic hepatitis C virus infection

doi:10.4254/wjh.v8.i25.1067

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World Journal of Hepatology

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World J Hepatol. Sep 8, 2016; 8(25): 1067-1074
Published online Sep 8, 2016. doi: 10.4254/wjh.v8.i25.1067

Table 2 Daily dietary intake of the chronic hepatitis C patients classified by cluster of differentiation 36 genotype

		CD36 genotype
Variable	Reference values	AA	AG	GG	P-value
Total calories	-	2531.3 ± 301.3	1902.5 ± 396.1	1873.5 ± 345.7	0.021^a
CH (%)	50-60	55.4 ± 10.5	54.3 ± 8.9	53.2 ± 6.4	0.76
Protein (%)	15	17.2 ± 4.6	16.3 ± 3.9	16.4 ± 2.9	0.81
Fat (%)	< 30	34.9 ± 7.5	27.5 ± 7.2	24.9 ± 1.1	0.001299^a
SFA (%)	< 7	16.1 ± 6.1	8.1 ± 3.2	8.4 ± 2.7	0.2 × 10^-6^a
MUFA (%)	20	13.1 ± 3.4	12.8 ± 7.6	12.1 ± 5.4	0.94
PUFA (%)	10	8.8 ± 6.5	5.6 ± 4.2	5.2 ± 1.3	0.11

^aBy post hoc tests: Total calories: AA genotype vs AG and GG genotypes, P = 0.027. Fat: AA vs AG, P = 0.006; AA vs GG, P = 0.002; SFA: AA vs AG, P = 0.2 × 10^-6, AA vs GG, P = 0.185 × 10^-4. Quantitative values are expressed as mean ± SD. CH: Carbohydrates; SFA: Saturated fatty acids; MUFA: Monounsaturated fatty acids; PUFAs: Polyunsaturated fatty acids; CD36: Cluster of differentiation 36.

Citation: Ramos-Lopez O, Roman S, Martinez-Lopez E, Fierro NA, Gonzalez-Aldaco K, Jose-Abrego A, Panduro A. CD36 genetic variation, fat intake and liver fibrosis in chronic hepatitis C virus infection. World J Hepatol 2016; 8(25): 1067-1074
URL: https://www.wjgnet.com/1948-5182/full/v8/i25/1067.htm
DOI: https://dx.doi.org/10.4254/wjh.v8.i25.1067