Copyright
©The Author(s) 2015.
World J Hepatol. May 8, 2015; 7(7): 1007-1011
Published online May 8, 2015. doi: 10.4254/wjh.v7.i7.1007
Published online May 8, 2015. doi: 10.4254/wjh.v7.i7.1007
Table 2 Etiology of hyperammonemia of non-hepatic origin[21]
Age | Category | Examples |
Adults | Stressful events | - |
Partial enzyme deficiency | - | |
Infection | Urinary tract infections (urease-producing organism, such as proteus mirabilis) | |
Pediatrics | Medications | Valproate |
Topiramate | ||
Salicylates such as aspirin | ||
Acetazolamide | ||
Zonisamide | ||
High-dose chemotherapy (5-fluorouracil) | ||
Infection | Urinary tract infections (urease-producing organism, such as proteus mirabilis) | |
Organic acidemias | Isovalericacidemia, propionic acidemia, methylmalonicacidemia, glutaricacidemia type II, multiple carboxylase deficiency, beta-ketothiolase deficiency | |
Congenital lactic acidosis | Pyruvate dehydrogenase deficiency | |
Pyruvate carboxylase deficiency | ||
Mitochondrial disorders | ||
Fatty acid oxidation defects | Acyl CoA dehydrogenase deficiency | |
Systemic carnitine deficiency | ||
Dibasic aminoacid transport defects | Lysinuric protein intolerance | |
Hyperammonemia-hyperornithinemia-homocitrullinuria | ||
Miscellaneous | Transient hyperammonemia of the newborn asphyxia | |
Reye syndrome | ||
Lactic acidosis |
- Citation: Kromas ML, Mousa OY, John S. Hyperammonemia-induced encephalopathy: A rare devastating complication of bariatric surgery. World J Hepatol 2015; 7(7): 1007-1011
- URL: https://www.wjgnet.com/1948-5182/full/v7/i7/1007.htm
- DOI: https://dx.doi.org/10.4254/wjh.v7.i7.1007