Focal liver lesions: Practical magnetic resonance imaging approach

Figure 3 Hepatocellular adenomas. In- (A) and opposed-phase (B) GRE T1-WI, fat-suppressed FSE T2-WI (C), pre (D) and post hepatocyte-specific contrast agent (Eovist^®) fat-suppressed 3D-GRE T1-WI at the arterial (E), portal venous (F), interstitial (G) and hepatobiliary (H) phases. Two focal liver lesions are noted on the left and caudate lobes (arrows, A-G) of a noncirrhotic liver, showing slight drop of signal intensity on opposed-phase (B) comparing with the in-phase (A) T1-WI, which is related to minimally fat content. Note that the liver parenchyma also shows minimal steatosis. The lesions demonstrate mild high signal intensity on T2-WI (C), heterogeneous enhancement on the postcontrast arterial phase image (E) and subsequent washout on later postcontrast images (F and G). On the hepatobiliary phase, acquired 20 min after the hepatocyte-specific contrast agent, the lesions show no contrast uptake, excluding the diagnosis of FNHs. GRE: Gradient-echo; FSE: Fast spinecho; T1-WI: T1-weighted images; FNH: Focal nodular hyperplasias.