Hepatic encephalopathy: An approach to its multiple pathophysiological features

doi:10.4254/wjh.v4.i3.50

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Mar 27, 2012 (publication date) through Nov 5, 2024

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World Journal of Hepatology

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1948-5182

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Baishideng Publishing Group Inc, 7041 Koll Center Parkway, Suite 160, Pleasanton, CA 94566, USA

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World J Hepatol. Mar 27, 2012; 4(3): 50-65
Published online Mar 27, 2012. doi: 10.4254/wjh.v4.i3.50

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Figure 3 Proposed sequences that finally induce changes in the long term potentials, which is down regulated in hyperammonemia (modified from Lynch[182]). AMPA: α-amino-3-hydroxy-5-methyl-4-isoxazolepropionic acid receptor; NMDA: N-metil D-aspartato; mGlu: Metabotropic glutamate receptors; PI-3K: Phosphatidylinositol 3-kinases; PKA and PKC: Protein kinase A and C; CaMKII: Ca²⁺/calmodulin-dependent protein kinases II.

Citation: Perazzo JC, Tallis S, Delfante A, Souto PA, Lemberg A, Eizayaga FX, Romay S. Hepatic encephalopathy: An approach to its multiple pathophysiological features. World J Hepatol 2012; 4(3): 50-65
URL: https://www.wjgnet.com/1948-5182/full/v4/i3/50.htm
DOI: https://dx.doi.org/10.4254/wjh.v4.i3.50