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©The Author(s) 2025.
World J Hepatol. Mar 27, 2025; 17(3): 101340
Published online Mar 27, 2025. doi: 10.4254/wjh.v17.i3.101340
Published online Mar 27, 2025. doi: 10.4254/wjh.v17.i3.101340
Figure 7 Western blot results of ionized calcium-binding adaptor molecule-1 and Takeda G protein-coupled receptor 5 protein expression in cerebral cortex of rats in each group (mean ± SD, n = 10).
A: Protein expression bands of ionized calcium-binding adaptor molecule-1 and Takeda G protein-coupled receptor 5 in the cerebral cortex of thioacetamide-induced liver cirrhosis model with GADPH as the upper sample control; B and C: The expression of ionized calcium-binding adaptor molecule-1 and Takeda G protein-coupled receptor 5 protein in each group, respectively. aP < 0.05 compared with vehicle + control diet group; bP < 0.05 compared with thioacetamide group. TAA: Thioacetamide; TGR5: Takeda G protein-coupled receptor 5; IBA-1: Ionized calcium-binding adaptor molecule-1.
- Citation: Ren C, Cha L, Huang SY, Bai GH, Li JH, Xiong X, Feng YX, Feng DP, Gao L, Li JY. Dysregulation of bile acid signal transduction causes neurological dysfunction in cirrhosis rats. World J Hepatol 2025; 17(3): 101340
- URL: https://www.wjgnet.com/1948-5182/full/v17/i3/101340.htm
- DOI: https://dx.doi.org/10.4254/wjh.v17.i3.101340