Chelation therapy in liver diseases of childhood: Current status and response

doi:10.4254/wjh.v13.i11.1552

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Nov 27, 2021 (publication date) through Dec 16, 2024

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World Journal of Hepatology

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1948-5182

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Baishideng Publishing Group Inc, 7041 Koll Center Parkway, Suite 160, Pleasanton, CA 94566, USA

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World J Hepatol. Nov 27, 2021; 13(11): 1552-1567
Published online Nov 27, 2021. doi: 10.4254/wjh.v13.i11.1552

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Figure 1 Pathophysiology of Wilson’s disease. Due to mutation in ATP 7B gene, P type ATPase is defective and copper is not incorporated in ceruloplasmin. Free copper increases in blood and is deposited in liver and extrahepatic sites (brain, kidneys, bones, cornea, RBC).

Citation: Seetharaman J, Sarma MS. Chelation therapy in liver diseases of childhood: Current status and response. World J Hepatol 2021; 13(11): 1552-1567
URL: https://www.wjgnet.com/1948-5182/full/v13/i11/1552.htm
DOI: https://dx.doi.org/10.4254/wjh.v13.i11.1552