Clinical Trials Study
Copyright ©The Author(s) 2020.
World J Hepatol. Nov 27, 2020; 12(11): 1076-1088
Published online Nov 27, 2020. doi: 10.4254/wjh.v12.i11.1076
Figure 1
Figure 1 Flowchart of patients and study design. HBeAg: Hepatitis B e antigen; CHB: Chronic hepatitis B; Peg-IFN: Pegylated interferon; NAs: Nucleos(t)ids analogues.
Figure 2
Figure 2 Hepatitis B surface antigen delta (Δ) (log10 IU/mL) at wk 24, 48 and 96 according to treatment group. Peg-IFN: Pegylated interferon; NA: Nucleos(t)ids analogue.
Figure 3
Figure 3 Hepatitis B surface antigen delta (Δ) (log10 IU/mL) according to interleukin 28B polymorphism and treatment group. A: Hepatitis B surface antigen delta (Δ) in interleukin 28B CC patients (n = 22); B: Hepatitis B surface antigen delta (Δ) in interleukin 28B CT/TT patients (n = 26). NS: Not significant; NA: Nucleos(t)ids analogue; Peg-IFN: Pegylated interferon.
Figure 4
Figure 4 Linear regression model of hepatitis B surface antigen levels according to treatment group. NA: Nucleos(t)ids analogue; Peg-IFN: Pegylated interferon; LR: Linear regression.
Figure 5
Figure 5 Rate of patients with low hepatitis B surface antigen levels (Hepatitis B surface antigen < 100 IU/mL and 100-1000 IU/mL) according to treatment group. A: NA group; B: Peg-IFN-NA group. NS: Not significant; NA: Nucleos(t)ids analogue; Peg-IFN: Pegylated interferon.