Amyotrophic lateral sclerosis as a protein level, non-genomic disease: Therapy with S2RM exosome released molecules

doi:10.4252/wjsc.v9.i11.187

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Nov 26, 2017 (publication date) through Jan 6, 2025

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World Journal of Stem Cells

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1948-0210

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Baishideng Publishing Group Inc, 7041 Koll Center Parkway, Suite 160, Pleasanton, CA 94566, USA

Therapeutics Advances

World J Stem Cells. Nov 26, 2017; 9(11): 187-202
Published online Nov 26, 2017. doi: 10.4252/wjsc.v9.i11.187

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Figure 2 Motor neuron in amyotrophic lateral sclerosis. ALS Motor neurons have misfolded SOD, reduced HSP and mitochondrial function. ALS: Amyotrophic lateral sclerosis; SOD: Superoxide dismutase; SOD1: Superoxide dismutase - misfolded; HSP: Heat shock proteins - disrupted ECM not permissive to HSP transfer; MTC: Mitochondria; ECM: Extracellular matrix.

Citation: Maguire G. Amyotrophic lateral sclerosis as a protein level, non-genomic disease: Therapy with S2RM exosome released molecules. World J Stem Cells 2017; 9(11): 187-202
URL: https://www.wjgnet.com/1948-0210/full/v9/i11/187.htm
DOI: https://dx.doi.org/10.4252/wjsc.v9.i11.187