Antralization at the edge of proximal gastric ulcers: Does Helicobacter pylori infection play a role?

doi:10.3748/wjg.v9.i6.1265

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Jun 15, 2003 (publication date) through Nov 5, 2024

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World Journal of Gastroenterology

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1007-9327

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Baishideng Publishing Group Inc, 7041 Koll Center Parkway, Suite 160, Pleasanton, CA 94566, USA

H. Pylori

World J Gastroenterol. Jun 15, 2003; 9(6): 1265-1269
Published online Jun 15, 2003. doi: 10.3748/wjg.v9.i6.1265

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Figure 3 Gastric mucosa at the ulcer edge before and after eradication of H. pylori infection in the same patient. A, biopsy of the ulcer edge before H. pylori eradication showing antral-type gastric mucosa with severe active chronic inflammation; B, biopsy of the healed ulcer site after H. pylori eradication showing body-type gastric mucosa with presence of parietal and chief cells in the gastric glands and mild residual chronic inflammation. Haematoxylin & eosin (H&E) staining × 250.

Citation: Xia HHX, Lam SK, Wong WM, Hu WHC, Lai KC, Wong SH, Leung SY, Yuen ST, Wright NA, Wong BCY. Antralization at the edge of proximal gastric ulcers: Does Helicobacter pylori infection play a role? World J Gastroenterol 2003; 9(6): 1265-1269
URL: https://www.wjgnet.com/1007-9327/full/v9/i6/1265.htm
DOI: https://dx.doi.org/10.3748/wjg.v9.i6.1265