Role of activation-induced cell death in pathogenesis of patients with chronic hepatitis B

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Oct 15, 2003 (publication date) through Dec 25, 2024

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World Journal of Gastroenterology

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1007-9327

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Baishideng Publishing Group Inc, 7041 Koll Center Parkway, Suite 160, Pleasanton, CA 94566, USA

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World J Gastroenterol. Oct 15, 2003; 9(10): 2356-2358
Published online Oct 15, 2003. doi: 10.3748/wjg.v9.i10.2356

Table 2 The density of IFN-γ and IL-10 from medium of each group (x^-±s, pg·mL^-1)

Group	n	IFN-γ	IL-10
Patient with induction	26	728.32 ± 149.59^a^,^b^,^c	175.75 ± 34.65^d
Patient without induction	26	1313.35 ± 403.98^b	74.48 ± 37.21
Healthy control with induction	15	2255.18 ± 465.56^c	188.86 ± 66.26
Healthy control without induction	15	2379.22 ± 465.33	190.58 ± 46.65

^aP < 0.01 (F = 7.37) vs. other three groups;

^bP < 0.01 (F = 232.94) vs. healthy control groups;

^cP < 0.01 (F = 25.90) vs. groups without induction;

^dP > 0.05 (F = 0.02) vs. other three groups.

Citation: Hou CS, Wang GQ, Lu SL, Yue B, Li MR, Wang XY, Yu JW. Role of activation-induced cell death in pathogenesis of patients with chronic hepatitis B. World J Gastroenterol 2003; 9(10): 2356-2358
URL: https://www.wjgnet.com/1007-9327/full/v9/i10/2356.htm
DOI: https://dx.doi.org/10.3748/wjg.v9.i10.2356