Copyright
©The Author(s) 2003.
World J Gastroenterol. Jan 15, 2003; 9(1): 54-58
Published online Jan 15, 2003. doi: 10.3748/wjg.v9.i1.54
Published online Jan 15, 2003. doi: 10.3748/wjg.v9.i1.54
Table 1 Expressions of c-myc, bcl-2, p53, Rb and hTERTmRNA in the different gastric diseases (%)
| GC | IMI-II | IMIII | DysI-II | DysIII | GC | |
| c-myc | 5 (11.90) | 8 (17.39) | 11 (44.00) | 22 (45.83) | 16 (59.26) | 74 (88.09) |
| bcl-2 | 3 (7.14) | 17 (36.96) | 10 (40.00) | 24 (50.00) | 18 (66.67) | 55 (65.48) |
| p53 | 0 (0.00) | 3 (6.52) | 5 (20.00) | 14 (29.17) | 10 (37.04) | 64 (76.19) |
| Rb | 3 (7.14) | 4 (8.70) | 8 (32.00) | 14 (29.17) | 12 (44.44) | 61 (72.62) |
| HTERTmRNA | 0 (0.00) | 1 (2.17) | 4 (16.00) | 10 (16.67) | 12 (44.44) | 78 (92.86) |
- Citation: Lan J, Xiong YY, Lin YX, Wang BC, Gong LL, Xu HS, Guo GS. Helicobacter pylori infection generated gastric cancer through p53-Rb tumor-suppressor system mutation and telomerase reactivation. World J Gastroenterol 2003; 9(1): 54-58
- URL: https://www.wjgnet.com/1007-9327/full/v9/i1/54.htm
- DOI: https://dx.doi.org/10.3748/wjg.v9.i1.54