LncRNA FTX promotes colorectal cancer radioresistance through disturbing redox balance and inhibiting ferroptosis via miR-625-5p/SCL7A11 axis

doi:10.3748/wjg.v31.i16.104305

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World Journal of Gastroenterology

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1007-9327

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Baishideng Publishing Group Inc, 7041 Koll Center Parkway, Suite 160, Pleasanton, CA 94566, USA

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World J Gastroenterol. Apr 28, 2025; 31(16): 104305
Published online Apr 28, 2025. doi: 10.3748/wjg.v31.i16.104305

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Figure 8 Molecular mechanism of lncRNA FTX in colorectal cancer radio resistance. LncRNA FTX/miR-625-5p regulated downstream molecule SLC7A11 pathway helps cancer cells facilitates the tolerance of ferroptosis and repair of DNA damage in colorectal cancer cells, which contributes to the transformation of colorectal cancer cells into radiotherapy-resistant cells. CRC: Colorectal cancer.

Citation: Dai Q, Qu TY, Yang JL, Leng J, Fang L, Zhu QQ, Wu KB, Wu J, Ma JJ, Yu HF. LncRNA FTX promotes colorectal cancer radioresistance through disturbing redox balance and inhibiting ferroptosis via miR-625-5p/SCL7A11 axis. World J Gastroenterol 2025; 31(16): 104305
URL: https://www.wjgnet.com/1007-9327/full/v31/i16/104305.htm
DOI: https://dx.doi.org/10.3748/wjg.v31.i16.104305