LncRNA FTX promotes colorectal cancer radioresistance through disturbing redox balance and inhibiting ferroptosis via miR-625-5p/SCL7A11 axis

doi:10.3748/wjg.v31.i16.104305

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Apr 28, 2025 (publication date) through Apr 27, 2025

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World Journal of Gastroenterology

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1007-9327

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Baishideng Publishing Group Inc, 7041 Koll Center Parkway, Suite 160, Pleasanton, CA 94566, USA

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World J Gastroenterol. Apr 28, 2025; 31(16): 104305
Published online Apr 28, 2025. doi: 10.3748/wjg.v31.i16.104305

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Figure 6 Overexpression of SLC7A11 promotes the repair of damaged DNA induced by ionizing radiation in colorectal cancer cells. A: Immunofluorescence assay detected γH2AX expression in HT29R-shFTX and HCT116R-shFTX cells after overexpression of SLC7A11 at 1 or 12 hours; B: Western blotting analyses determined the expression of mismatch repair proteins in HT29R-shFTX and HCT116R-shFTX cells after SLC7A11 overexpression. ^aP < 0.05 and ^bP < 0.01. NC: Negative control.

Citation: Dai Q, Qu TY, Yang JL, Leng J, Fang L, Zhu QQ, Wu KB, Wu J, Ma JJ, Yu HF. LncRNA FTX promotes colorectal cancer radioresistance through disturbing redox balance and inhibiting ferroptosis via miR-625-5p/SCL7A11 axis. World J Gastroenterol 2025; 31(16): 104305
URL: https://www.wjgnet.com/1007-9327/full/v31/i16/104305.htm
DOI: https://dx.doi.org/10.3748/wjg.v31.i16.104305