SEL1L-mediated endoplasmic reticulum associated degradation inhibition suppresses proliferation and migration in Huh7 hepatocellular carcinoma cells

Figure 7 The expression of EXT2 in human tissue and the survival analysis. A: Correlation analysis of EXT2 and SEL1L in liver hepatocellular carcinoma (LIHC) and liver tissues performed by GEPIA2; B: Correlation analysis of heavy-chain binding protein and SEL1L in LIHC and liver tissues performed by GEPIA2; The expression of EXT2 and SEL1L in patients in different subgroups were compared based on clinical factors such as C and D: Alcohol consumption; E and F: Neoplasm histologic grade; G and H: Hepatitis virus infection; I: Survival analysis of EXT2 in all patients; J and K: Survival analysis of EXT2 in patients with/without hepatitis virus infection. TPM: Transcripts per million; HBV: Hepatitis B virus; HCV: Hepatitis C virus; HR: Hazard ratio.