COVID-19, liver dysfunction and pathophysiology: A conceptual discussion

doi:10.3748/wjg.v28.i6.683

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World Journal of Gastroenterology

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1007-9327

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World J Gastroenterol. Feb 14, 2022; 28(6): 683-688
Published online Feb 14, 2022. doi: 10.3748/wjg.v28.i6.683

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Figure 1 Pathophysiological hypotheses explaining liver injury by severe acute respiratory syndrome coronavirus-2. ACE2: Angiotensin II; ADAM17: AT1R-metalloprotease 17; CD147: Cluster of Differentiation 147; L-SIGN: Liver/Lymph node-specific intercellular adhesion molecule-3-grabbing non-integrin.

Citation: Gonçalves Júnior J. COVID-19, liver dysfunction and pathophysiology: A conceptual discussion. World J Gastroenterol 2022; 28(6): 683-688
URL: https://www.wjgnet.com/1007-9327/full/v28/i6/683.htm
DOI: https://dx.doi.org/10.3748/wjg.v28.i6.683