Copyright
©The Author(s) 2021.
World J Gastroenterol. Jun 7, 2021; 27(21): 2850-2870
Published online Jun 7, 2021. doi: 10.3748/wjg.v27.i21.2850
Published online Jun 7, 2021. doi: 10.3748/wjg.v27.i21.2850
Tissue source | Non-COVID-19 autopsy samples | COVID-19 autopsy samples | Total number of analyzed samples |
Lung | H1Lu; H2Lu; H3Lu; H4Lu | Case1: Samples 1, 2, 3, 4; Case2: Samples 1, 2, 3; Case3: Samples 1, 2; Case4: Samples 1, 2; Case5: Samples 1, 2, 3, 4, 5; Case6: Samples 1, 2, 3, 4, 5; Case7: Samples 1, 2, 3, 4, 5; Case8: Samples 1, 2, 3, 4, 5; Case9: Samples 1, 2, 3, 4, 5; Case10: Samples 1, 2, 3; Case11: Samples 1, 2, 3 | 4 healthy samples and 42 COVID-19 samples |
Liver | H1Li; H2Li; H3Li; H4Li | Case3: Sample 1; Case4: Sample 1; Case5: Sample 1; Case8: Sample 1; Case10: Sample 1; Case12: Sample1 | 4 healthy samples and 6 COVID-19 samples |
- Citation: Hammoudeh SM, Hammoudeh AM, Bhamidimarri PM, Mahboub B, Halwani R, Hamid Q, Rahmani M, Hamoudi R. Insight into molecular mechanisms underlying hepatic dysfunction in severe COVID-19 patients using systems biology. World J Gastroenterol 2021; 27(21): 2850-2870
- URL: https://www.wjgnet.com/1007-9327/full/v27/i21/2850.htm
- DOI: https://dx.doi.org/10.3748/wjg.v27.i21.2850