Copyright
©The Author(s) 2020.
World J Gastroenterol. Feb 7, 2020; 26(5): 499-513
Published online Feb 7, 2020. doi: 10.3748/wjg.v26.i5.499
Published online Feb 7, 2020. doi: 10.3748/wjg.v26.i5.499
Figure 6 Rescue experiment.
A: miR-34c inhibitor could counteract the effect of si E2F1 on related proteins. aP < 0.05, and bP < 0.01; B: miR-34c inhibitor could offset the inhibitory effect of si E2F1 on cell activity. aP < 0.05 vs other groups; C: miR-34c inhibitor neutralized the sensitization effect of si-E2FL on cell resistance. bP < 0.01, and eP < 0.001; D: miR-34c inhibitor inhibited the effect of si-E2FL on apoptosis. aP < 0.05, bP < 0.01, and eP < 0.001; E: miR-34c inhibitor inhibited the inhibitory effect of si-E2FL on S-phase cells. bP < 0.01 and eP < 0.001.
- Citation: Zheng H, Wang JJ, Yang XR, Yu YL. Upregulation of miR-34c after silencing E2F transcription factor 1 inhibits paclitaxel combined with cisplatin resistance in gastric cancer cells. World J Gastroenterol 2020; 26(5): 499-513
- URL: https://www.wjgnet.com/1007-9327/full/v26/i5/499.htm
- DOI: https://dx.doi.org/10.3748/wjg.v26.i5.499