Copyright
©The Author(s) 2020.
World J Gastroenterol. Nov 7, 2020; 26(41): 6361-6377
Published online Nov 7, 2020. doi: 10.3748/wjg.v26.i41.6361
Published online Nov 7, 2020. doi: 10.3748/wjg.v26.i41.6361
Figure 1 Increased expression of P-selectin glycoprotein ligand 1 on neutrophils and monocytes from patients with acute pancreatitis.
A: Flow cytometry for CD14-positive cells to detect monocytes in the peripheral blood of patients (n = 21) with acute pancreatitis (AP) or normal control subjects (n = 8); B: Boxplot showing quantification of monocytes; C: Flow cytometry for CD33-positive cells to detect neutrophils in the peripheral blood of patients (n = 21) with AP or normal control subjects (n = 8); D: Boxplot showing quantification of neutrophils; E: Fluorescence intensity of P-selectin glycoprotein ligand 1 (PSGL-1) on neutrophils and monocytes; F: Boxplot showing quantification of fluorescence intensity of PSGL-1 on neutrophils and monocytes. aP < 0.05, Student's t-test. PSGL-1: P-selectin glycoprotein ligand 1.
- Citation: Zhang X, Zhu M, Jiang XL, Liu X, Liu X, Liu P, Wu XX, Yang ZW, Qin T. P-selectin glycoprotein ligand 1 deficiency prevents development of acute pancreatitis by attenuating leukocyte infiltration. World J Gastroenterol 2020; 26(41): 6361-6377
- URL: https://www.wjgnet.com/1007-9327/full/v26/i41/6361.htm
- DOI: https://dx.doi.org/10.3748/wjg.v26.i41.6361