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©The Author(s) 2020.
World J Gastroenterol. Oct 14, 2020; 26(38): 5822-5835
Published online Oct 14, 2020. doi: 10.3748/wjg.v26.i38.5822
Published online Oct 14, 2020. doi: 10.3748/wjg.v26.i38.5822
Figure 5 Acetyl-11-keto-β-boswellic acid inhibits the monolayer wound healing of gastric cancer cells.
A and B: Phase micrographs of BGC823 and SGC7901 cells at various times after monolayer wounding (original magnification, × 100); C and D: Quantification of cell migration using monolayer wound-healing assay. The gastric cancer cells were treated without (control) or with 20 µM acetyl-11-keto-β-boswellic acid. Each data point represents the mean ± SD from three independent experiments. aP < 0.05, bP < 0.01 vs control (0 µM). AKBA: Acetyl-11-keto-β-boswellic acid.
- Citation: Sun MX, He XP, Huang PY, Qi Q, Sun WH, Liu GS, Hua J. Acetyl-11-keto-β-boswellic acid inhibits proliferation and induces apoptosis of gastric cancer cells through the phosphatase and tensin homolog /Akt/ cyclooxygenase-2 signaling pathway. World J Gastroenterol 2020; 26(38): 5822-5835
- URL: https://www.wjgnet.com/1007-9327/full/v26/i38/5822.htm
- DOI: https://dx.doi.org/10.3748/wjg.v26.i38.5822