MiR-32-5p aggravates intestinal epithelial cell injury in pediatric enteritis induced by Helicobacter pylori

Figure 1 Aberrant expression of miR-32-5p in enteritis regulates biological function of intestinal epithelial cells. A: Expression of miR-32-5p in serum of pediatric enteritis; B: Expression of miR-32-5p in intestinal epithelial cells infected by Helicobacter pylori (H. pylori); C: Cell viability measurement in intestinal epithelial cells transfected with miR-32-5p inhibitor in the presence or absence of H. pylori; D: Viability measurement in intestinal epithelial cells transfected with miR-32-5p mimic in the presence or absence of H. pylori; E: Apoptosis of intestinal epithelial cells with miR-32-5p inhibitor transfection in the presence or absence of H. pylori; F: Apoptosis of intestinal epithelial cells with miR-32-5p mimic transfection in the presence or absence of H. pylori; G: The mRNA level of TNF-α in H. pylori-infected intestinal epithelial cells in the presence of miR-32-5p inhibitor; I: The mRNA level of TNF-α in H. pylori-infected intestinal epithelial cells in the presence of miR-32-5p mimic; H: The mRNA level of IL-6 in H. pylori-infected intestinal epithelial cells in the presence of miR-32-5p inhibitor; J: The mRNA level of IL-6 in H. pylori-infected intestinal epithelial cells in the presence of miR-32-5p mimic. ^bP < 0.01. H. pylori: Helicobacter pylori.